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or catalyzed by glutathione peroxidase (GSH-Px) to produce H 2 O: 2GSH+H 2 O 2 →2H 2 O+GSSG, all of which complete the antioxidant scavenging process and constitute the antioxidant enzymatic response system of the organism, maintaining the intracellular redox balance ( 2+ and Cu + are capable of redox reactions with unpaired electrons, destroying the structure and function of proteins, nucleic acids and lipids and cross-linking with these macromolecules to produce toxic substances ( We know that mitochondria are the main site of ROS production and a target organelle for oxidative stress ( 1 F 0 ATP synthase to produce ATP for OXPHOS ( 2 being produced in the tricarboxylic acid (TCA) cycle, and too many electron donors entering the ETC, leading to a maximum mitochondrial voltage gradient ( 2 .- , which exceeds the antioxidant scavenging capacity and leads to oxidative stress, causing impairment of cellular respiratory function, promoting apoptosis and ultimately leading to dysfunction of diseased tissues and systems ( Figure 2 )