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Normal mitochondrial function reduces myocardial oxidative damage, while abnormal mitochondrial function can cause chronic myocardial hypoxia and exacerbate myocardial fibrotic degeneration by impairing respiratory function
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As the most abundant isoform of the GPX family, GPX1 exerts its impacts via regulating gene expression, protein function, and enzyme activities of key factors involved in both macro- and micro-nutrient metabolism [97, 122, 123]
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