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According to the obtained results, curcumin carriers activate the Th1 response by elevating INF-γ and suppressing the Th2 response by reducing IL-4, which is a profit for an anti-tumor drug in cancer therapy
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Further investigation into how post-stroke sympathetic hyperactivity triggers myocardial injury–particularly through region-specific brain lesions and downstream catecholamine-mediated signaling pathways–remains a critical research priority with profound clinical implications for precision prevention and targeted therapies
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