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When cysteinesulfinate itself was used as substrate, isolated hepatocytes, renal cortical tubules, enterocytes, perfused isolated hindquarter, and intact rats all exhibited a high capacity for cysteinesulfinate metabolism to carbon dioxide or sulfate, with rates of cysteinesulfinate oxidation far exceeding those for Cys catabolism to (Bagley and Stipanuk 1995
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Proinflammatory responses of heme in alveolar macrophages: repercussion in lung hemorrhagic episodes
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