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If this delay were to prevent fork damage in pol delta mutant cells, the inhibition of Chk1 would not affect cell viability
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Indeed ROS species can serve as signal mediators in the redox regulation of cell metabolism [19], as they are enzymatically produced and degraded by NADPH-oxidases, which generate superoxide anions [3], SOD, which generates H 2 O 2 , and catalase and peroxidase, which detoxify H 2 O 2 into H 2 O
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