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In conclusion, we generated and characterized a novel rat model that recapitulates the phenotypes in TSHR Y444X mutation in human, which will also help to study GPCR related signaling during development and the pathomechanism of CH
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Ran-GST pull-down assays using various PlexinB1 deletion constructs demonstrated that the Rho binding domain (RBD) of PlexinB1 is required for the interaction of Ran with PlexinB1 (Supplementary Fig
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