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Among the NLGN isoforms, NLGN1 is predominantly involved in excitatory synapse structure, transmission, and plasticity, but has minimal effects on inhibitory synapses.While its direct link to ASD remains unclear, alterations in NLGN1 expression may contribute to cognitive impairments through disruptions in synaptic plasticity and learning-related neural circuits (82)

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A mitochondrial impairment, due to increasing oxidative stress, has also been described in CFTR-knockout mice where both an oxidative damage to mtDNA and a reduced aconitase activity have been observed [121]